Brown Bowel Disease
Excess fish oil can paint the intestines black, and melanin is not the culprit.
It can even happen all the way from the front end to the rear end (stem to stern).
The culprits are omega-3 fatty acids, especially the ones from algae and fish oil.
Brown Bowel Syndrome (intestinal lipofuscinosis) is supposed to be a “rare” condition.
Don’t bet on it.
Vitamin E offers limited protection against Yellow Fat Disease and its partner in crime, Brown Bowel Syndrome.
So do tiny doses of selenium.
So do dozens of toxic chemicals, and, of course, the global food cartels use them extensively because they’re cheaper and more profitable to manufacture than vitamin E.
Petra M. Burgmann, D.V.M. (Feeding Your Pet Bird, 1993) wrote …
“Vitamin E is not a single compound, but actually a group of compounds called tocopherols, all with similar vitamin E-like activity. Of these, alpha tocopherol has the greatest activity. There are also many synthetic chemical compounds with similar chemical structures and a number of chemicals that are unrelated chemically to the tocopherols that also have vitamin E-like activity.”
According to Fenoglio-Preiser’s Gastrointestinal Pathology, Fourth Edition, 2017 …
“Patients with vitamin E deficiency develop eosinophilic enteritis and brown bowel syndrome. Vitamin E deficiency occurs alone or it complicates other diseases. Patients range in age from the 20s to the late 70s, with an average age of 51 years. They present with epigastric pain, mild diarrhea, and chronic malabsorption. Mitochondrial alterations may be the source of the lipofuscin pigment, and therefore, the term smooth muscle mitochondrial myopathy may apply to the disorder.
“Grossly, the bowel is variably orange-brown and is often retrospectively described as being darker than usual by the surgeon. The segmental or diffuse brownish discoloration can be appreciated from the serosal aspect of the GI tract as well as on cut section. The disorder more commonly affects the small intestine and stomach, but it can involve the colon. Occasionally, it involves the entire GI tract. No correlation exists between the degree of pigmentation and the severity of the associated disease.
“The mucosa usually appears normal. However, occasionally the villi appear blunted, and there may be mild submucosal edema and submucosal or mural eosinophilic inflammation. A coarsely autofluorescent, granular, golden brown pigment, known as lipofuscin, fills the smooth muscle cells of the muscularis propria, muscularis mucosae, and vascular walls, as well as macrophages. The pigment appears round to oval, often lying in a perinuclear or central cellular location.”
Golly, I wonder how lipofuscin pigment invaded the gut in the first place?
Could it be something we ate?